[1] Qin XH, Li YB, He ML, et al. Folic acid therapy reduces
serum uric acid in hypertensive patients: a substudy of the
China Stroke Primary Prevention Trial (CSPPT)[J]. Am J Clin
Nutr, 2017, 105(4):882-889.
[2] Marcucci R, Betti I, Cecchi E, et al. Hyperhomocysteinemia
and vitamin B6 deficiency: new risk markers for nonvalvular
atrial fibrillation[J]. Am Heart J, 2004, 148(3):456-461.
[3] Ohishi T, Fujita T, Suzuki D, et al. Serum homocysteine
levels are affected by renal function during a 3-year period of
minodronate therapy in female osteoporotic patients[J]. J Bone
Miner Metab, 2019, 37(2):319-326.
[4] Liu KJ, Xuekelati SYE, Zhou K, et al. Expression profiles
of six atherosclerosis-associated microRNAs that cluster in
patients with hyperhomocysteinemia: a clinical study[J]. DNA
Cell Biol, 2018, 37(3):189-198.
[5] Peng HY, Man CF, Xu J, et al. Elevated homocysteine levels
and risk of cardiovascular and all-cause mortality: a metaanalysis
of prospective studies[J]. J Zhejiang Univ Sci B,
2015, 16(1):78-86.
[6] Serapinas D, Boreikaite E, Bartkeviciute A, et al. The
importance of folate, vitamins B6 and B12 for the lowering
of homocysteine concentrations for patients with recurrent
pregnancy loss and MTHFR mutations[J]. Reprod Toxicol,
2017, 72:159-163.
[7] Zhang Z, Wei C, Zhou Y, et al. Homocysteine induces
apoptosis of human umbilical vein endothelial cells via
mitochondrial dysfunction and endoplasmic reticulum
stress[J]. Oxid Med Cell Longev, 2017, 2017:5736506.
[8] Wu X, Zhang LH, Miao Y, et al. Homocysteine causes
vascular endothelial dysfunction by disrupting endoplasmic
reticulum redox homeostasis[J]. Redox Biol, 2019, 20:46-59.
[9] Baszczuk A, KopczyńskiZ, Thielemann A. Endothelial
dysfunction in patients with primary hypertension and
hyperhomocysteinemia[J]. Postepy Hig Med Dosw (Online),
2014, 68:91-100.
[10] Balint B, Jepchumba VK, Guéant JL et al. Mechanisms of
homocysteine-induced damage to the endothelial, medial and
adventitial layers of the arterial wall[J]. Biochimie. 2020,
173:100-106.
[11] Zhang LH, Li ZL, Xing CC, et al. The protective mechanism
of folic acid on hyperhomocysteinemia-related arterial injury
in spontaneously hypertensive rats: folic acid against arterial
inflammation[J]. Vascular, 2022, 30(5):988-998.
[12] Kaplan P, Tatarkova Z, Sivonova MK, et al. Homocysteine
and mitochondria in cardiovascular and cerebrovascular
systems[J]. Int J Mol Sci. 2020, 21(20):7698.
[13] Sandberg K, Ji H, Hay M. Sex-specific immune modulation of
primary hypertension[J]. Cell Immunol, 2015, 294(2):95-101.
[14] Sowmya S, Swathi Y, Yeo AL, et al. Hydrogen sulfide:
regulatory role on blood pressure in hyperhomocysteinemia[J].
Vascul Pharmacol, 2010, 53(3/4):138-143.
[15] Shi L, Liu XY, Huang ZG, et al. Endogenous Hydrogen sulfide
and ERK1/2-STAT3 signaling pathway may participate in
the association between homocysteine and hypertension[J]. J
Geriatr Cardiol, 2019, 16(11):822-834.
[16] Lei XW, Li Q, Zhang JZ, et al. The protective roles of
folic acid in preventing diabetic retinopathy are potentially
associated with suppressions on angiogenesis, inflammation,
and oxidative stress[J]. Ophthalmic Res, 2019, 62(2):80-92.
[17] Christen WG, Cook NR, Van Denburgh M, et al. Effect
of combined treatment with folic acid, vitamin B6, and
vitamin B12 on plasma biomarkers of inflammation and
endothelial dysfunction in women[J]. J Am Heart Assoc, 2018,
7(11):e008517.