索引超出了数组界限。 文章摘要
|本期目录/Table of Contents|

[1]周子颖,唐其柱.受体相互作用蛋白 2 与心血管疾病[J].国际心血管病杂志,2023,02:85-87.
点击复制

受体相互作用蛋白 2 与心血管疾病(PDF)

《国际心血管病杂志》[ISSN:1006-6977/CN:61-1281/TN]

期数:
2023年02期
页码:
85-87
栏目:
综述
出版日期:
2023-03-20

文章信息/Info

Title:
-
作者:
周子颖; 唐其柱
430060 武汉大学人民医院心血管内科代谢与相关慢病湖北省重点实验室
Author(s):
-
关键词:
受体相互作用蛋白2 ; 炎性反应; 动脉粥样硬化; 心肌梗死; 心肌重构
Keywords:
-
分类号:
-
DOI:
10.3969/j.issn.1673-6583.2023.02.006
文献标识码:
-
摘要:
受体相互作用蛋白2 是1 种激酶，可通过激活核因子κB 及丝裂原活化蛋白激 酶等信号通路参与动脉粥样硬化斑块的形成，影响心肌梗死后心功能，调控心肌重构进程。 该文介绍受体相互作用蛋白2 的结构、功能及其在心血管相关疾病中的作用。
Abstract:
-

参考文献/References

[1] Inohara N, del Peso L, Koseki T, et al. RICK, a novel protein kinase containing a caspase recruitment domain, interacts with CLARP and regulates CD95-mediated apoptosis[J]. J Biol Chem, 1998, 273(20):12296-12300.
[2] Gong Q, Long ZQ, Zhong FL, et al. Structural basis of RIP2 activation and signaling[J]. Nat Commun, 2018, 9(1):4993.
[3] Pellegrini E, Desfosses A, Wallmann A, et al. RIP2 filament formation is required for NOD2 dependent NF-κB signalling[J]. Nat Commun, 2018, 9(1):4043.
[4] Shehat MG, Cardona OA, Aranjuez GF, et al. RIP2 promotes FcγR-mediated reactive Oxygen species production[J]. J Biol Chem, 2019, 294(26):10365-10378.
[5] Olona A, Leishman S, Anand PK. The NLRP3 inflammasome: regulation by metabolic signals[J]. Trends Immunol, 2022, 43(12):978-989.
[6] Huang B, Gui M, Ni Z, et al. Chemotherapeutic drugs induce different gut microbiota disorder pattern and NOD/RIP2/NF- κB signaling pathway activation that lead to different degrees of intestinal injury[J]. MicrobiolSpectr, 2022, 10(6):e0167722.
[7] Rahman MA, Sundaram K, Mitra S, et al. Receptor interacting protein-2 plays a critical role in human lung epithelial cells survival in response to Fas-induced cell-death[J]. PLoS One, 2014, 9(3):e92731.
[8] Kisiswa L, Fernández-Suárez D, Sergaki MC, et al. RIP2 gates TRAF6 interaction with death receptor p75NTR to regulate cerebellar granule neuron survival[J]. Cell Rep, 2018, 24(4):1013-1024.
[9] Homer CR, Kabi A, Marina-García N, et al. A dual role for receptor-interacting protein kinase 2 (RIP2) kinase activity in nucleotide-binding oligomerization domain 2 (NOD2)- dependent autophagy[J]. J Biol Chem, 2012, 287(30):25565- 25576.
[10] Shao Y, Saredy J, Yang WY, et al. Vascular endothelial cells and innate immunity[J]. Arterioscler Thromb Vasc Biol, 2020, 40(6):e138-e152.
[11] Moreno L, McMaster SK, Gatheral T, et al. Nucleotide oligomerization domain 1 is a dominant pathway for NOS2 induction in vascular smooth muscle cells: comparison with Toll-like receptor 4 responses in macrophages[J]. Br J Pharmacol, 2010, 160(8):1997-2007.
[12] Reed DM, Foldes G, Gatheral T, et al. Pathogen sensing pathways in human embryonic stem cell derived-endothelial cells: role of NOD1 receptors[J]. PLoS One, 2014, 9(4):e91119.
[13] González-Ramos S, Paz-García M, Rius C, et al. Endothelial NOD1 directs myeloid cell recruitment in atherosclerosis through VCAM-1[J]. FASEB J, 2019, 33(3):3912-3921.
[14] Suebsuwong C, Dai B, Pinkas DM, et al. Receptorinteracting protein kinase 2 (RIPK2) and nucleotide-binding oligomerization domain (NOD) cell signaling inhibitors based on a 3,5-diphenyl-2-aminopyridine scaffold[J]. Eur J Med Chem, 2020, 200:112417.
[15] Jacquet S, Nishino Y, Kumphune S, et al. The role of RIP2 in p38 MAPK activation in the stressed heart[J]. J Biol Chem, 2008, 283(18):11964-11971.
[16] Sicard P, Jacquet S, Kobayashi KS, et al. Pharmacological postconditioning effect of muramyl dipeptide is mediated through RIP2 and TAK1[J]. Cardiovasc Res, 2009, 83(2):277- 284.
[17] Andersson L, ScharinT?ng M, Lundqvist A, et al. Rip2 modifies VEGF-induced signalling and vascular permeability in myocardial ischaemia[J]. Cardiovasc Res, 2015, 107(4):478- 486.
[18] Li LP, Wang XD, Chen W, et al. Regulatory role of CARD3 in left ventricular remodelling and dysfunction after myocardial infarction[J]. Basic Res Cardiol, 2015, 110(6):56.
[19] Zhao CH, Ma X, Guo HY, et al. RIP2 deficiency attenuates cardiac hypertrophy, inflammation and fibrosis in pressure overload induced mice[J]. Biochem Biophys Res Commun, 2017, 493(2):1151-1158.
[20] Lin HB, Naito K, Oh Y, et al. Innate immune nod1/ RIP2 signaling is essential for cardiac hypertrophy but requires mitochondrial antiviral signaling protein for signal transductions and energy balance[J]. Circulation, 2020, 142(23):2240-2258.

备注/Memo

备注/Memo:
通信作者：唐其柱， E-mail：qztang@whu.edu.cn
更新日期/Last Update: 2023-03-20