索引超出了数组界限。 文章摘要
|本期目录/Table of Contents|

[1]王茜.硫化氢在心肌缺血再灌注损伤中的作用机制[J].国际心血管病杂志,2021,04:200-202.
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硫化氢在心肌缺血再灌注损伤中的作用机制(PDF)

《国际心血管病杂志》[ISSN:1006-6977/CN:61-1281/TN]

期数:
2021年04期
页码:
200-202
栏目:
综述
出版日期:
2021-08-15

文章信息/Info

Title:
-
作者:
王茜
200040 上海市药品和医疗器械不良反应监测中心
Author(s):
-
关键词:
硫化氢 心肌缺血再灌注损伤 炎性反应 细胞凋亡
Keywords:
-
分类号:
-
DOI:
10.3969/j.issn.1673-6583.2021.04.003
文献标识码:
-
摘要:
硫化氢(H2S)是一种气体信号分子,在哺乳动物体内广泛存在。内源性H2S主要在线粒体中代谢。外源性或内源性H2S对心肌缺血再灌注损伤均具有保护作用,主要作用机制包括抗炎、抗氧化、抗凋亡和促进血管新生等。该文介绍H2S在心肌缺血再灌注损伤中的作用和机制及H2S供体相关药物,以期对缺血性心脏病的干预提供新思路。
Abstract:
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参考文献/References

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备注/Memo

备注/Memo:
通信作者:王茜,E-mail:wangqianlily@126.com
更新日期/Last Update: 2021-08-15