索引超出了数组界限。
[1] Abraham WT, Fisher WG, Smith AL,et al. Cardiac resychronization in chronic heart failure [J]. N Engl J Med,2002,346(24):1845-1853.
[2] Singh JP, Klein HU, Huang DT, et al. Left ventricular lead position and clinical outcome in the multicenter automatic defibrillator implantation trial-cardiac resynchronization therapy(MADIT-CRT)trial [J]. Circulation,2011,123(11):1159-1166.
[3] Leyva F, Nisam S, Auricchio A, et al.20 Years of Cardiac Resynchronization Therapy [J]. J Am Coll Cardiol,2014,64(10):1047-1058.
[4] Brignole M, Auricchio A, Baron-Esquivias G, et al. 2013 ESC Guidelines on cardiac pacing and cardiac resynchronization therapy [J]. Eur Heart J,2013,34(29):2281-2329.
[5] Chung ES, Leon AR, Tavazzi L, et al. Results of the Predictors of Response to CRT(PROSPECT)trial [J]. Circulation,2008,117(20):2608-2616.
[6] 陆秋芬,李毅刚,俞洁霏,等.心力衰竭患者心脏再同步化疗效的多因素分析 [J].国际心血管病杂志,2014,41(1):58-60.
[7] Ypenburg C, van Bommel RJ, Delgado V, et al. Optimal left ventricular lead position predicts reverse remodeling and survival after cardiac resynchronization therapy [J]. J Am Coll Cardiol, 2008,52(17):1402-1409.
[8] Lambiase PD, Rinaldi A, Hauck J, et al. Non-contact left ventricular endocardial mapping in cardiac resynchronisation therapy [J]. Heart,2004,90(1):44-51.
[9] Lindenfeld J, Feldman AM, Saxon L, et al. Effects of cardiac resynchronization therapy with or without a defibrillator on survival and hospitalizations in patients with New York Heart Association class IV heart failure [J]. Circulation,2007,115(2):204-212.
[10] Kandala J, Upadhyay GA, Altman RK, et al. QRS morphology, left ventricular lead location, and clinical outcome in patients receiving cardiac resynchronization therapy [J]. Eur Heart J,2013,34(29):2252-2262.
[11] Khan FZ, Virdee MS, Fynn SP, et al. Left ventricular lead placement in cardiac resynchronization therapy: where and how? [J]. Europace,2009,11(5):554-561.
[12] Yu CM, Fang JW,Zhang Q,et al. Tissue Doppler imaging is superior to strain rate imaging and postsystolic shortening on the prediction of reversere modeling in both isehemie and nonischemic heart failure after cardiac resynehronization therapy [J]. Circulation.,2004,110(1):66-73.
[13] Bilchick KC, Kuruvilla S, Hamirani YS, et al. Impact of mechanical activation, scar, and electrical timing on cardiac resynchronization therapy response and clinical outcomes [J]. J Am Coll Cardiol, 2014,63(16):1657-1666.
[14] Bilchick KC, Dimaano V, Wu KC, et al. Cardiac magnetic resonance assessment of dyssynchrony and myocardial scar predicts function class improvement following cardiac resynchronization therapy [J]. JACC Cardiovasc. Imaging, 2008,1(5):561-568.
[15] Woo GW, Petersen-stejskal S, Johnson JW, et al. Ventricular reverse remodeling and 6-month outcomes in patients receiving CRT:analysis of MIRACLE study [J]. J Interv Card Electrophysiol, 2005,12(2):107-113.
[16] Wikstrom G, Blomström-Lundqvist C, Andren B, et al. The effects of aetiology on outcome in patients treated with cardiac resynchronization therapy in the CARE-HF trial [J]. Eur Heart J, 2009,30(7):782-788.
[17] Chalil S, Foley PW, Muyhaldeen SA, et al. Late gadolinium enhancement-cardiovascular magnetic resonance as a predictor of response to cardiac resynchronization therapy in patients with ischaemic cardiomyopathy [J]. Europace, 2007,9(11):1031-1037.
[18] Ypenburg C, Roes SD, Bleeker GB, et al. Effect of total scar burden on contrast-enhanced magnetic resonance imaging on response to cardiac resynchronization therapy [J]. Am J Cardiol,2007,99(5):657-660.
[19] Hummel JP Lindner JR, Belcik JT, et al. Extent of myocardial viability predicts response to biventricular pacing in ischemic cardiomyopathy [J]. Heart Rhythm,2005,2(11):1211-1217.
[20] Ypenburg C,Schalij MJ, Bleeker GB, et al. Impact of viability and scar tissue on response to cardiac resynchronization therapy in ischaemic heart failure patients [J]. Eur Heart J, 2007,28(1):33-41.