索引超出了数组界限。
[1] Liu MX, Li W, Wang HB, et al. CTRP9 ameliorates atrial inflammation, fibrosis, and vulnerability to atrial fibrillation in post-myocardial infarction rats[J]. J Am Heart Assoc, 2019, 8(21):e013133.
[2] Liu Z, Finet JE, Wolfram JA, et al. Calcium/calmodulin-dependent protein kinase Ⅱ causes atrial structural remodeling associated with atrial fibrillation and heart failure[J]. Heart Rhythm, 2019, 16(7):1080-1088.
[3] Liao J, Wu QF, Qian C, et al. TRPV4 blockade suppresses atrial fibrillation in sterile pericarditis rats[J]. JCI Insight, 2020, 5(23):e137528.
[4] Zhou HS, Peng DW, Lai YY, et al. Activation of PKCα participates in the reduction of Ikur in atrial myocytes induced by tumour necrosis factor-α[J]. Clin Exp Pharmacol Physiol, 2020, 48(3):435-442.
[5] Lazzerini PE, Laghi-Pasini F, Acampa M, et al. Systemic inflammation rapidly induces reversible atrial electrical remodeling: the role of interleukin-6-mediated changes in connexin expression[J]. J Am Heart Assoc, 2019, 8(16):e011006.
[6] Narducci ML, Pelargonio G, Russo AD, et al. Role of tissue C-reactive protein in atrial cardiomyocytes of patients undergoing catheter ablation of atrial fibrillation: pathogenetic implications[J]. Europace, 2011, 13(8):1133-1140.
[7] Jiang ZQ, Dai LD, Song ZY, et al. Association between C-reactive protein and atrial fibrillation recurrence after catheter ablation: a meta-analysis[J]. Clin Cardiol, 2013, 36(9):548-554.
[8] Amdur R, Mukherjee M, Go A, et al. Interleukin-6 is a risk factor for atrial fibrillation in chronic kidney disease: findings from the CRIC study[J]. PLoS One, 2016, 11(2):e0148189.
[9] 戴艳枝, 王晓华, 刘秋玲, 等. 白细胞介素-1β、白细胞介素-8及肿瘤坏死因子-α与房颤伴凝血功能异常的相关性[J].中国当代医药, 2019, 26(23):4-7.
[10] Matsushita N, Ishida N, Ibi M, et al. IL-1β Plays an important role in pressure overload-induced atrial fibrillation in mice[J]. Biol Pharm Bull, 2019, 42(4):543-546.
[11] Silva R. Influence of inflammation and atherosclerosis in atrial fibrillation[J]. Curr Atheroscler Rep, 2017, 19(1):2.
[12] Luan Y, Guo YY, Li SF, et al. Interleukin-18 among atrial fibrillation patients in the absence of structural heart disease[J]. Europace, 2010, 12(12):1713-1718.
[13] Damås JK, Aukrust P, Ueland T, et al. Monocyte chemoattractant protein-1 enhances and interleukin-10 suppresses the production of inflammatory cytokines in adult rat cardiomyocytes[J]. Basic Res Cardiol, 2001, 96(4):345-352.
[14] Takahashi N, Kume O, Wakisaka O, et al. Novel strategy to prevent atrial fibrosis and fibrillation[J]. Circ J, 2012, 76(10):2318-2326.
[15] Zhang GG, Zhang L, Zhou XH, et al. Monocyte chemoattractant protein-1-induced protein in age-related atrial fibrillation and its association with circulating fibrosis biomarkers[J]. Cardiology, 2019, 142(4):244-249.
[16] Bonaterra GA, Zügel S, Thogersen J, et al. Growth differentiation factor-15 deficiency inhibits atherosclerosis progression by regulating interleukin-6-dependent inflammatory response to vascular injury[J]. J Am Heart Assoc, 2012, 1(6):e002550.
[17] Wallentin L, Hijazi Z, Andersson U, et al. Growth differentiation factor 15, a marker of oxidative stress and inflammation, for risk assessment in patients with atrial fibrillation: insights from the Apixaban for Reduction in Stroke and Other Thromboembolic Events in Atrial Fibrillation(ARISTOTLE)trial[J]. Circulation, 2014, 130(21):1847-1858.
[18] Berg DD, Ruff CT, Jarolim P, et al. Performance of the ABC scores for assessing the risk of stroke or systemic embolism and bleeding in patients with atrial fibrillation in ENGAGE AF-TIMI 48[J]. Circulation, 2019, 139(6):760-771.
[19] Hu YF, Chen YJ, Lin YJ, et al. Inflammation and the pathogenesis of atrial fibrillation[J]. Nat Rev Cardiol, 2015, 12(4):230-243.
[20] Rudolph V, Andrié RP, Rudolph TK, et al. Myeloperoxidase acts as a profibrotic mediator of atrial fibrillation[J]. Nat Med, 2010, 16(4):470-474.
[21] Wang PY, Cheng M, Wang PX, et al. SNP rs2243828 in MPO associated with myeloperoxidase level and atrial fibrillation risk in Chinese Han population[J]. J Cell Mol Med, 2020, 24(17):10263-10266.
[22] Tan Q, Zhang SY, Zou XY, et al. Fluvastatin therapy could not decrease progression of paroxysmal atrial fibrillation in non-valvular disease patients[J]. Anatol J Cardiol, 2017, 18(2):103-107.
[23] Zhen- Han L, Rui S, Dan C, et al. Perioperative statin administration with decreased risk of postoperative atrial fibrillation, but not acute kidney injury or myocardial infarction: a meta-analysis[J]. Sci Rep, 2017, 7(1):10091.
[24] Darghosian L, Free M, Li J, et al. Effect of omega-three polyunsaturated fatty acids on inflammation, oxidative stress, and recurrence of atrial fibrillation[J]. Am J Cardiol, 2015, 115(2):196-201.
[25] Ozaydin M, Erdogˇan D, Tayyar S, et al. N-3 polyunsaturated fatty acids administration does not reduce the recurrence rates of atrial fibrillation and inflammation after electrical cardioversion: a prospective randomized study[J]. Anadolu Kardiyol Derg, 2011, 11(4):305-309.
[26] Rodrigo R, Korantzopoulos P, Cereceda M, et al. A randomized controlled trial to prevent post-operative atrial fibrillation by antioxidant reinforcement[J]. J Am Coll Cardiol, 2013, 62(16):1457-1465.
[27] Liu C, Wang JH, Yiu DF, et al. The efficacy of glucocorticoids for the prevention of atrial fibrillation, or length of intensive care unite or hospital stay after cardiac surgery: a meta-analysis[J]. Cardiovasc Ther, 2014, 32(3):89-96.
[28] Stavrakis S, Humphrey MB, Scherlag BJ, et al. Low-level transcutaneous electrical vagus nerve stimulation suppresses atrial fibrillation[J]. J Am Coll Cardiol, 2015, 65(9):867-875.
[29] Stavrakis S, Humphrey MB, Scherlag BJ, et al. Low-level vagus nerve stimulation suppresses post-operative atrial fibrillation and inflammation: a randomized study[J]. JACC Clin Electrophysiol, 2017, 3(9):929-938.