索引超出了数组界限。
[1] Cui Y, Chen LJ, Huang T, et al. The pharmacology, toxicology
and therapeutic potential of anthraquinone derivative
emodin[J]. Chin J Nat Med, 2020, 18(6):425-435.
[2] Li QQ, Gao J, Pang XH, et al. Molecular mechanisms of
action of emodin: as an anti-cardiovascular disease drug[J].
Front Pharmacol, 2020, 11:559607.
[3] Luo N, Fang J, Wei LQ, et al. Emodin in atherosclerosis
prevention: pharmacological actions and therapeutic
potential[J]. Eur J Pharmacol, 2021, 890:173617.
[4] Pang XM, Liu JT, Li YX, et al. Emodin inhibits homocysteineinduced
C-reactive protein generation in vascular smooth muscle
cells by regulating PPARγ expression and ROS-ERK1/2/p38
signal pathway[J]. PLoS One, 2015, 10(7): e0131295.
[5] 白岩, 苏振丽, 孙瀚麒, 等. 芦荟大黄素对高脂血症大鼠心肌
炎作用的研究[J]. 哈尔滨医科大学学报, 2018, 52(1):11-14.
[6] 王立民, 刘有旺, 王恩波, 等. 以大黄素为声敏剂介导声动
力疗法对小鼠动脉粥样硬化治疗作用的研究[J]. 医学信息,
2018, 31(6):58-60.
[7] Shou XL, Zhou RF, Zhu LY, et al. Emodin, a Chinese herbal
medicine, inhibits reoxygenation-induced injury in cultured
human aortic endothelial cells by regulating the peroxisome
Proliferator-Activated receptor-γ (PPAR-γ) and endothelial
nitric oxide synthase (eNOS) signaling pathway[J]. Med Sci
Monit, 2018, 24:643-651.
[8] Ye BZ, Chen XD, Dai SS, et al. Emodin alleviates myocardial
ischemia/reperfusion injury by inhibiting gasdermin
D-mediated pyroptosis in cardiomyocytes[J]. Drug Des Devel
Ther, 2019, 13:975-990.
[9] Zhang XZ, Qin QJ, Dai HY, et al. Emodin protects H9c2
cells from hypoxia-induced injury by up-regulating miR-138
expression[J]. Braz J Med Biol Res, 2019, 52(3):e7994.
[10] Li Y, Xu QQ, Shan CS, et al. Combined use of emodin and
ginsenoside Rb1 exerts synergistic neuroprotection in cerebral
ischemia/reperfusion rats[J]. Front Pharmacol, 2018, 9:943.
[11] Wang YQ, Liu Q, Cai JY, et al. Emodin prevents renal
ischemia-reperfusion injury via suppression of CAMKII/
DRP1-mediated mitochondrial fission[J]. Eur J Pharmacol,
2022, 916:174603.
[12] 祁蕾, 苑博, 傅强. 缺氧/再复氧与脂多糖激活肠上皮细胞核
转录因子-κB和低氧诱导因子-1α信号通路以及大黄素对其
的干预作用[J]. 中华危重病急救医学, 2014, 26(6):409-414.
[13] Evans LW, Bender A, Burnett L, et al. Emodin and emodinrich
rhubarb inhibits histone deacetylase (HDAC) activity
and cardiac myocyte hypertrophy[J]. J Nutr Biochem, 2020,
79:108339.
[14] Evans L, Shen Y, Bender A, et al. Divergent and overlapping
roles for selected phytochemicals in the regulation of
pathological cardiac hypertrophy[J]. Molecules, 2021, 26(5):
1210.
[15] Gao J, Zhang KL, Wang Y, et al. A machine learning-driven
study indicates emodin improves cardiac hypertrophy by
modulation of mitochondrial SIRT3 signaling[J]. Pharmacol
Res, 2020, 155:104739.
[16] Guan RJ, Wang X, Zhao XM, et al. Emodin ameliorates
bleomycin-induced pulmonary fibrosis in rats by suppressing
epithelial-mesenchymal transition and fibroblast activation[J].
Sci Rep, 2016, 6:35696.
[17] Xiao D, Zhang Y, Wang R, et al. Emodin alleviates cardiac
fibrosis by suppressing activation of cardiac fibroblasts via
upregulating metastasis associated protein 3[J]. Acta Pharm
Sin B, 2019, 9(4):724-733.
[18] Zhang HM, Wang FP, Qiu Y, et al. Emodin inhibits
coxsackievirus B3 replication via multiple signalling cascades
leading to suppression of translation[J]. Biochem J, 2016,
473(4):473-485.
[19] 吴常裕, 杨海燕. 大黄素对病毒性心肌炎患者的疗效及血清
指标的影响[J]. 临床药物治疗杂志, 2018, 16(9):39-42.
[20] Li HM, Liu X, Meng ZY, et al. Kanglexin delays heart aging
by promoting mitophagy[J]. Acta Pharmacol Sin, 2022, 43(3):
613-623.