索引超出了数组界限。 文章摘要
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[1]刘芳,蒋庆庆,戴朝晖.高迁移率族蛋白1与心力衰竭[J].国际心血管病杂志,2020,06:348.
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高迁移率族蛋白1与心力衰竭(PDF)

《国际心血管病杂志》[ISSN:1006-6977/CN:61-1281/TN]

期数:
2020年06期
页码:
348
栏目:
综述
出版日期:
2020-12-15

文章信息/Info

Title:
-
作者:
刘芳; 蒋庆庆; 戴朝晖
410006 长沙市第四医院心血管内科
Author(s):
-
关键词:
高迁移率族蛋白1;  心力衰竭;  双向调控
Keywords:
-
分类号:
-
DOI:
10.3969/j.issn.1673-6583.2020.06.008
文献标识码:
A
摘要:
高迁移率族蛋白1(HMGB1)在机体受到病原体刺激、组织损伤后,经主动分泌及被动释放而迁移至细胞外,诱导各种相关炎性反应发生。研究证明HMGB1与心力衰竭的发病、病情进展和预后相关,在疾病进展中起到双向调控作用。
Abstract:
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参考文献/References

[1] Churchill M, Klass J, Zoetewey DL. Structural analysis of HMGD-DNA complexes reveals influence of intercalation on sequence selectivity and DNA bending[J]. J Mol Biol, 2010, 403(1):88-102.
[2] Joshi SR, Sarpong YC, Peterson RC, et al. Nucleosome dynamics: HMGB1 relaxes canonical nucleosome structure to facilitate estrogen receptor binding[J]. Nucleic Acids Res, 2012, 40(20):10161-10171.
[3] Lee SA, Kwak MS, Kim S, et al. The role of high mobility group box 1 in innate immunity[J]. Yonsei Med J, 2014, 55(5):1165-1176.
[4] Merkle T, Grasser KD. Unexpected mobility of plant chromatin-associated HMGB proteins[J]. Plant Signal Behav, 2011, 6(6):878-880.
[5] Mandke P, Vasquez KM. Interactions of high mobility group box protein 1(HMGB1)with nucleic acids: implications in DNA repair and immune responses[J]. DNA Repair(Amst), 2019, 83:102701.
[6] 于鹏, 姜红. 高迁移率族蛋白1与心血管疾病关系概述[J]. 中国分子心脏病学杂志, 2013, 13(2):509-512.
[7] Kang R, Chen R, Zhang Q, et al. HMGB1 in health and disease[J]. Mol Aspects Med, 2014, 40:1-116.
[8] Qu L, Chen C, Chen YY, et al. High-mobility group box 1(HMGB1)and autophagy in acute lung injury(ALI): a review[J]. Med Sci Monit, 2019, 25:1828-1837.
[9] Wang LJ, Lu L, Zhang FR, et al. Increased serum high-mobility group box-1 and cleaved receptor for advanced glycation endproducts levels and decreased endogenous secretory receptor for advanced glycation endproducts levels in diabetic and non-diabetic patients with heart failure[J]. Eur J Heart Fail, 2011, 13(4):440-449.
[10] Volz HC, Laohachewin D, Schellberg D, et al. HMGB1 is an independent predictor of death and heart transplantation in heart failure[J]. Clin Res Cardiol, 2012, 101(6):427-435.
[11] 武芬芬, 傅晓敏, 何昆仑. 高迁移率族蛋白 B1及内脂素在心肌梗死中的临床意义[J]. 河北医药, 2015, 37(17):2589-2592.
[12] 柴熙晨, 王玲洁, 苏倩, 等. 血清中高迁移率族蛋白B1水平与慢性心力衰竭患者预后的关系[J].内科理论与实践, 2012, 7(5):366-369.
[13] Raucci A, Capogrossi MC. Nuclear Hmgb1: the fix for the failing heart[J]. JACC Basic Transl Sci, 2019, 4(2):248-250.
[14] 张慧, 郗爱旗. 慢性心力衰竭患者体内HMGB1与sRAGE水平变化[J]. 世界最新医学信息文摘, 2018, 18(88):68-69.
[15] 丁华胜, 杨俊. 高迁移率族蛋白B1在心血管系统疾病中的作用[J]. 临床心血管病杂志, 2010, 26(4):244-246.
[16] Yang H, Wang H, Gzura CJ, et al. The cytokine activity of HMGB1[J]. J Leukoc Biol, 2005, 78(1):1-8.
[17] Funayama A, Shishido T, Netsu S, et al. Cardiac nuclear high mobility group box 1 prevents the development of cardiac hypertrophy and heart failure[J]. Cardiovasc Res, 2013, 99(4):657-664.
[18] Takahashi T, Shishido T, Kinoshita D, et al. Cardiac nuclear high-mobility group box 1 ameliorates pathological cardiac hypertrophy by inhibiting DNA damage response[J]. JACC Basic Transl Sci, 2019, 4(2):234-247.
[19] Ouyang F, Huang H, Zhang M, et al. HMGB1 induces apoptosis and EMT in association with increased autophagy following H/R injury in cardiomyocytes[J]. Int J Mol Med, 2016, 37(3):679-689.
[20] Zhang L, Liu M, Jiang H, et al. Extracellular high-mobility group box 1 mediates pressure overload-induced cardiac hypertrophy and heart failure[J]. J Cell Mol Med, 2016, 20(3):459-470.
[21] Foglio E, Puddighinu G, Germani A, et al. HMGB1 inhibits apoptosis following MI and induces autophagy via mTORC1 inhibition[J]. J Cell Physiol, 2017, 232(5):1135-1143.
[22] Takahashi K, Fukushima S, Yamahara K, et al. Modulated inflammation by injection of high-mobility group box 1 recovers post-infarction chronically failing heart[J]. Circulation, 2008, 118(14 Suppl):S106-S114.
[23] Limana F, Germani A, Zacheo A, et al. Exogenous high-mobility group box 1 protein induces myocardial regeneration after infarction via enhanced cardiac C-kit+ cell proliferation and differentiation[J]. Circ Res, 2005, 97(8):e73-e83.
[24] Kaneko M, Shintani Y, Narita T, et al. Extracellular high mobility group box 1 plays a role in the effect of bone marrow mononuclear cell transplantation for heart failure[J]. PLoS One, 2013, 8(10):e76908.
[25] Kohno T, Anzai T, Naito K, et al. Role of high-mobility group box 1 protein in post-infarction healing process and left ventricular remodeling[J]. Cardiovasc Res, 2009, 81(3):565-573.
[26] Oozawa S, Mori S, Kanke T, et al. Effects of HMGB1 on ischemia-reperfusion injury in the rat heart[J]. Circ J, 2008, 72(7):1178-1184.
[27] Andrassy M, Volz HC, Igwe JC, et al. High-mobility group box-1 in ischemia-reperfusion injury of the heart[J]. Circulation, 2008, 117(25):3216-3226.
[28] 张庆成, 张向峰, 毕春辉, 等. HMGB1和pro-BNP在心力衰竭进展期中的动态变化[J]. 中国医药科学, 2017, 7(1):188-190.
[29] 王丹. 冠心病PCI术患者HMGB1、BNP水平变化及与发生心力衰竭的关系[J]. 河北医药, 2019, 41(2):199-202.
[30] Tzeng HP, Fan J, Vallejo JG, et al. Negative inotropic effects of high-mobility group box 1 protein in isolated contracting cardiac myocytes[J]. Am J Physiol Heart Circ Physiol, 2008, 294(3):H1490-H1496.
[31] Ishii J, Cui W, Kitagawa F, et al. Prognostic value of combination of cardiac troponin T and B-type natriuretic peptide after initiation of treatment in patients with chronic heart failure[J]. Clin Chem, 2003, 49(12):2020-2026.
[32] Liu T, Zhang DY, Zhou YH, et al. Increased serum HMGB1 level may predict the fatal outcomes in patients with chronic heart failure[J]. Int J Cardiol, 2015, 184:318-320.
[33] 李霞, 张维龙. HMGB1对老年急性心力衰竭患者预后的评估价值[J]. 医学研究杂志, 2018, 47(8):180-183.
[34] Dong LY, Chen F, Xu M, et al. Quercetin attenuates myocardial ischemia-reperfusion injury via downregulation of the HMGB1-TLR4-NF-κB signaling pathway[J]. Am J Transl Res, 2018, 10(5):1273-1283.
[35] Marsh AM, Nguyen AH, Parker TM, et al. Clinical use of high mobility group box 1 and the receptor for advanced glycation end products in the prognosis and risk stratification of heart failure: a literature review[J]. Can J Physiol Pharmacol, 2017, 95(3):253-259.

备注/Memo

备注/Memo:
基金项目:湖南省卫计委科研课题A类项目(A2016017)通信作者:戴朝晖,E-mail:1987049780@qq.com
更新日期/Last Update: 2020-12-15