索引超出了数组界限。 文章摘要
|本期目录/Table of Contents|

[1]唐子健,李树仁,许文华,等.非阻塞性冠状动脉疾病与心外膜脂肪组织[J].国际心血管病杂志,2020,06:335.
点击复制

非阻塞性冠状动脉疾病与心外膜脂肪组织(PDF)

《国际心血管病杂志》[ISSN:1006-6977/CN:61-1281/TN]

期数:
2020年06期
页码:
335
栏目:
综述
出版日期:
2020-12-15

文章信息/Info

Title:
-
作者:
唐子健李树仁许文华申泽雪
050017 石家庄,河北医科大学研究生院(唐子健); 050051 石家庄,河北省人民医院心内科(李树仁); 063210 唐山,华北理工大学研究生院(许文华,申泽雪)
Author(s):
-
关键词:
非阻塞性冠状动脉疾病 心外膜脂肪组织 危险因素
Keywords:
-
分类号:
-
DOI:
10.3969/j.issn.1673-6583.2020.06.005
文献标识码:
A
摘要:
心外膜脂肪组织(EAT)参与非阻塞性冠状动脉疾病的发生发展过程,功能异常的EAT可导致心外膜血管及微血管功能障碍、冠状动脉痉挛、非阻塞性高危斑块形成及破裂,临床表现为非阻塞性冠状动脉疾病。针对EAT的监测及治疗,可能成为非阻塞性冠状动脉疾病诊治的新靶点。该文介绍非阻塞性冠状动脉疾病与EAT之间的关系。
Abstract:
-

参考文献/References

[1] Bairey Merz CN, Pepine CJ, Walsh MN. Ischemia and no obstructive coronary artery disease(INOCA)developing evidence-based therapies and research agenda for the next decade[J]. Circulation, 2017, 135(11):1075-1092.
[2] Dreyer RP, Tavella R, Curtis JP, et al. Myocardial infarction with non-obstructive coronary arteries as compared with myocardial infarction and obstructive coronary disease: outcomes in a medicare population[J]. Eur Heart J, 2020, 41(7):870-878.
[3] Crea F, Montone RA, Niccoli G. Myocardial infarction with non-obstructive coronary arteries: dealing with pears and apples[J]. Eur Heart J, 2020, 41(7):879-881.
[4] Neeland IJ, Ross R, Després JP, et al. Visceral and ectopic fat, atherosclerosis, and cardiometabolic disease: a position statement[J]. Lancet Diabetes Endocrinol, 2019, 7(9):715-725.
[5] 胡成平, 赵迎新. 心外膜脂肪组织与冠心病的关系[J]. 中华心血管病杂志, 2017, 45(5):447-450.
[6] Matloch Z, Cinkajzlova A, Mraz M, et al. The role of inflammation in epicardial adipose tissue in heart diseases[J]. Curr Pharm Des, 2018, 24(3):297-309.
[7] Iacobellis G. Local and systemic effects of the multifaceted epicardial adipose tissue depot[J]. Nat Rev Endocrinol, 2015, 11(6):363-371.
[8] Iozzo P. Myocardial, perivascular, and epicardial fat[J]. Diabetes Care, 2011, Suppl 2:S371-S379.
[9] Sara JDS, Prasad M, Zhang M, et al. High-sensitivity C-reactive protein is an independent marker of abnormal coronary vasoreactivity in patients with non-obstructive coronary artery disease[J]. Am Heart J, 2017, 190:1-11.
[10] Prasad M, Matteson EL, Herrmann J, et al. Uric acid is associated with inflammation, coronary microvascular dysfunction, and adverse outcomes in postmenopausal women[J]. Hypertension, 2017, 69(2):236-242.
[11] 廖玉华, 余淼, 史河水. 心外膜脂肪组织:心血管病防治的新靶点[J]. 临床心血管病杂志, 2020, 36(1):11-14.
[12] Vaccarino V, Bremner JD. Behavioral, emotional and neurobiological determinants of coronary heart disease risk in women[J]. Neurosci Biobehav Rev, 2017, 74(Pt B):297-309.
[13] Daniel M, Agewall S, Berglund F, et al. Prevalence of anxiety and depression symptoms in patients with myocardial infarction with non-obstructive coronary arteries[J]. Am J Med, 2018, 131(9):1118-1124.
[14] Hung MY, Mao CT, Hung MJ, et al. Coronary artery spasm as related to anxiety and depression: a nationwide population-based study[J]. Psychosom Med, 2019, 81(3):237-245.
[15] Kahl KG, Hueper K, Schweiger U, et al. Pericardial, intra-abdominal, and subcutaneous adipose tissue in patients with major depressive disorder[J]. Acta Psychiatr Scand, 2014, 130(2):137-143.
[16] Amigues I, Russo C, Giles JT, et al. Myocardial microvascular dysfunction in rheumatoid arthritis[J]. Circ Cardiovasc Imaging, 2019, 12(1):e30636512.
[17] Fatma E, Bunyamin K, Savas S, et al. Epicardial fat thickness in patients with rheumatoid arthritis[J]. Afr Health Sci, 2015, 15(2):489-495.
[18] Sade LE, Eroglu S, Bozba H, et al. Relation between epicardial fat thickness and coronary flow reserve in women with chest pain and angiographically normal coronary arteries[J]. Atherosclerosis, 2009, 204(2):580-585.
[19] 伍琼, 杨波. 心外膜脂肪垫厚度与非阻塞性冠状动脉慢血流的关系[J]. 中华心血管病杂志, 2016, 44(11):956-960.
[20] Chang TY, Hsu CY, Chiu CC, et al. Association between echocardiographic epicardial fat thickness and circulating endothelial progenitor cell level in patients with stable angina pectoris[J]. Clin Cardiol, 2017, 40(9):697-703.
[21] Jia G, Aroor AR, Sowers JR. The role of mineralocorticoid receptor signaling in the cross-talk between adipose tissue and the vascular wall[J]. Cardiovasc Res, 2017, 113(9):1055-1063.
[22] Guzik TJ, Skiba DS, Touyz RM, et al. The role of infiltrating immune cells in dysfunctional adipose tissue[J]. Cardiovasc Res, 2017, 113(9):1009-1023.
[23] Nosalski R, Guzik TJ. Perivascular adipose tissue inflammation in vascular disease[J]. Br J Pharmacol, 2017, 174(20):3496-3513.
[24] Nishio S, Kusunose K, Yamada H, et al. Echocardiographic epicardial adipose tissue thickness is associated with symptomatic coronary vasospasm during provocative testing[J]. J Am Soc Echocardiogr, 2017, 30(10):1021-1027.
[25] Ohyama K, Matsumoto Y, Takanami K, et al. Coronary adventitial and perivascular adipose tissue inflammation in patient with vasospastic angina[J]. J Am Coll Cardiol, 2018, 71(4):414-425.
[26] Chang L, Xiong W, Zhao XJ, et al. Bmal1 in perivascular adipose tissue regulates resting-phase blood pressure through transcriptional regulation of angiotensinogen[J]. Circulation, 2018, 138(1):67-79.
[27] Xia N, Li HG. The role of perivascular adipose tissue in obesity-induced vascular dysfunction[J]. Br J Pharmacol, 2017, 174(20):3425-3442.
[28] Park JS, Choi SY, Zheng M, et al. Epicardial adipose tissue thickness is a predictor for plaque vulnerability in patients with significant coronary artery disease[J]. Atherosclerosis, 2013, 226(1):134-139.
[29] Tan Y, Zhou J, Zhou Y, et al. Epicardial adipose tissue is associated with high-risk plaque feature progression in non-culprit lesions[J]. Int J Cardiovasc Imaging, 2017, 33(12):2029-2037.
[30] Kaji H. Adipose tissue-derived plasminogen activator inhibitor-1 function and regulation[J]. Compr Physiol, 2016, 6(4):1873-1896.
[31] Vilahur G, Ben-Aicha S, Badimon L. New insights into the role of adipose tissue in thrombosis[J]. Cardiovasc Res, 2017, 113(9):1046-1054.
[32] Gaibazzi N, Martini C, Botti A, et al. Coronary inflammation by computed tomography pericoronary fat attenuation in MINOCA and Tako-Tsubo syndrome[J]. J Am Heart Assoc, 2019, 8(17):e013235.
[33] Packer M. Drugs that ameliorate epicardial adipose tissue inflammation may have discordant effects in heart failure with a preserved ejection fraction as compared with a reduced ejection fraction[J]. J Card Fail, 2019, 25(12):986-1003.
[34] Mckenney-Drake ML, Rodenbeck SD, Bruning RS, et al. Epicardial adipose tissue removal potentiates outward remodeling and arrests coronary atherogenesis[J]. Ann Thorac Surg, 2017, 103(5):1622-1630.

备注/Memo

备注/Memo:
通信作者:李树仁,E-mail:lsr64@126.com
更新日期/Last Update: 2020-12-15