索引超出了数组界限。
[1] Davies MJ. Stability and instability: two faces of coronary atherosclerosis. The Paul Dudley White Lecture 1995[J]. Circulation, 1996, 94(8):2013-2020.
[2] Nicholls SJ, Ballantyne CM, Barter PJ, et al. Effect of two intensive statin regimens on progression of coronary disease[J]. N Engl J Med, 2011, 365(22):2078-2087.
[3] Diamantis E, Kyriakos G, Quiles-Sanchez LV, et al. The anti-inflammatory effects of statins on coronary artery disease: an updated review of the literature[J]. Curr Cardiol Rev, 2017, 13(3):209-216.
[4] Ridker PM. The Jupiter trial: results, controversies, and implications for prevention[J]. Circ Cardiovasc Qual Outcomes, 2009, 2(3):279-285.
[5] Puri R, Nissen SE, Libby P, et al. C-reactive protein, but not low-density lipoprotein cholesterol levels, associate with coronary atheroma regression and cardiovascular events after maximally intensive statin therapy[J]. Circulation, 2013, 128(22):2395-2403.
[6] Nakagomi A, Shibui T, Kohashi K, et al. Differential effects of atorvastatin and pitavastatin on inflammation, insulin resistance, and the carotid Intima-Media thickness in patients with dyslipidemia[J]. J Atheroscler Thromb, 2015, 22(11):1158-1171.
[7] Khurana S, Gupta S, Bhalla H, et al. Comparison of anti-inflammatory effect of atorvastatin with rosuvastatin in patients of acute coronary syndrome[J]. J Pharmacol Pharmacother, 2015, 6(3):130-135.
[8] Lagace TA, Curtis DE, Garuti R, et al. Secreted PCSK9 decreases the number of LDL receptors in hepatocytes and in livers of parabiotic mice[J]. J Clin Invest, 2006, 116(11):2995-3005.
[9] Everett BM, Smith RJ, Hiatt WR. Reducing LDL with PCSK9 inhibitors—the clinical benefit of lipid drugs[J]. N Engl J Med, 2015, 373(17):1588-1591.
[10] Nicholls SJ, Puri R, Anderson T, et al. Effect of evolocumab on progression of coronary disease in statin-treated patients: the GLAGOV randomized clinical trial[J]. JAMA, 2016, 316(22):2373-2384.
[11] Su G, Sun G, Liu H, et al. Niacin suppresses progression of atherosclerosis by inhibiting vascular inflammation and apoptosis of vascular smooth muscle cells[J]. Med Sci Monit, 2015, 21:4081-4089.
[12] Capra V, Bäck M, Angiolillo DJ, et al. Impact of vascular thromboxane prostanoid receptor activation on hemostasis, thrombosis, oxidative stress, and inflammation[J]. J Thromb Haemost, 2014, 12(2):126-137.
[13] Gremmel T, Koppensteiner R, Kaider A, et al. Impact of variables of the P-selectin—P-selectin glycoprotein ligand-1 axis on leukocyte-platelet interactions in cardiovascular disease[J]. Thromb Haemost, 2015, 113(4):806-812.
[14] Mezouar S, Darbousset R, Dignat-George F, et al. Inhibition of platelet activation prevents the P-selectin and integrin-dependent accumulation of cancer cell microparticles and reduces tumor growth and metastasis in vivo[J]. Int J Cancer, 2015, 136(2):462-475.
[15] Takeda M, Yamashita T, Shinohara M, et al. Beneficial effect of anti-platelet therapies on atherosclerotic lesion formation assessed by phase-contrast X-ray CT imaging[J]. Int J Cardiovasc Imaging, 2012, 28(5):1181-1191.
[16] Dobesh PP, Patel M. The Parthenon clinical development program: the role of ticagrelor in patients with atherothrombotic disease[J]. Cardiovasc Drugs Ther, 2017, 31(4):433-444.
[17] Preusch MR, Rusnak J, Staudacher K, et al. Ticagrelor promotes atherosclerotic plaque stability in a mouse model of advanced atherosclerosis[J]. Drug Des Devel Ther, 2016, 10:2691-2699.
[18] Heart Outcomes Prevention Evaluation Study Investigators, Yusuf S, Sleight P, et al. Effects of an angiotensin-converting-enzyme inhibitor, ramipril, on cardiovascular events in high-risk patients[J]. N Engl J Med, 2000, 342(3):145-153.
[19] Mason RP. Optimal therapeutic strategy for treating patients with hypertension and atherosclerosis: focus on olmesartan medoxomil[J]. Vasc Health Risk Manag, 2011, 7:405-416.
[20] Zinellu A, Sotgia S, Mangoni AA, et al. Effects of ramipril and telmisartan on plasma concentrations of low molecular weight and protein thiols and carotid intima media thickness in patients with chronic kidney disease[J]. Dis Markers, 2016, 2016:1821596.
[21] Bäck M, Hansson GK. Leukotriene receptors in atherosclerosis[J]. Ann Med, 2006, 38(7):493-502.
[22] Ingelsson E, Yin L, Bäck M. Nationwide cohort study of the leukotriene receptor antagonist montelukast and incident or recurrent cardiovascular disease[J]. J Allergy Clin Immunol, 2012, 129(3):702-707.
[23] Fisk M, Gajendragadkar PR, Mäki-Petäjä KM, et al. Therapeutic potential of p38 MAP kinase inhibition in the management of cardiovascular disease[J]. Am J Cardiovasc Drugs, 2014, 14(3):155-165.
[24] Sarov-Blat L, Morgan JM, Fernandez P, et al. Inhibition of p38 mitogen-activated protein kinase reduces inflammation after coronary vascular injury in humans[J]. Arterioscler Thromb Vasc Biol, 2010, 30(11):2256-2263.
[25] Sharma PA, Maheshwari R, Tekade M, et al. Nanomaterial based approaches for the diagnosis and therapy of cardiovascular diseases[J]. Curr Pharm Des, 2015, 21(30):4465-4478.
[26] Tang J, Lobatto ME, Hassing L, et al. Inhibiting macrophage proliferation suppresses atherosclerotic plaque inflammation[J]. Sci Adv, 2015, 1(3):e1400223.
[27] Zhang J, Nie S, Martinez-Zaguilan R, et al. Formulation, characteristics and antiatherogenic bioactivities of CD36-targeted epigallocatechin gallate(EGCG)-loaded nanoparticles[J]. J Nutr Biochem, 2016, 30:14-23.
[28] Fredman G, Kamaly N, Spolitu S, et al. Targeted nanoparticles containing the proresolving peptide Ac2-26 protect against advanced atherosclerosis in hypercholesterolemic mice [J]. Sci Transl Med, 2015, 7(275):275ra20.
[29] Kimura T, Tse K, Sette A, et al. Vaccination to modulate atherosclerosis[J]. Autoimmunity, 2015, 48(3):152-160.
[30] Chyu KY, Dimayuga PC, Shah PK. Vaccine against arteriosclerosis: an update[J]. Ther Adv Vaccines, 2017, 5(2):39-47.