索引超出了数组界限。 文章摘要
|本期目录/Table of Contents|

[1]赵健斌,吴登轩,陈世群,等.对比剂诱导急性肾损伤的机制与预防策略[J].国际心血管病杂志,2016,05:280-282,285.
点击复制

对比剂诱导急性肾损伤的机制与预防策略(PDF)

《国际心血管病杂志》[ISSN:1006-6977/CN:61-1281/TN]

期数:
2016年05期
页码:
280-282,285
栏目:
综述
出版日期:
2016-09-20

文章信息/Info

Title:
-
作者:
赵健斌; 吴登轩; 陈世群; 刘勇; 谭宁; 陈纪言
510080 广东省人民医院心内科,广东省医学科学院,广东省心管病研究所,广东省冠心病重点实验室
Author(s):
-
关键词:
碘对比剂; 细胞毒性; 黏度; 急性肾损伤; 水化
Keywords:
-
分类号:
-
DOI:
10.3969/j.issn.1673-6583.2016.05.007
文献标识码:
-
摘要:
对比剂诱导的急性肾损伤(CIAKI)是使用碘对比剂后较常见的并发症。碘对比剂的细胞毒性是CIAKI发病的触发因子,能直接损伤肾小管上皮细胞和血管内皮细胞,最终导致肾髓质缺氧。而碘对比剂的高黏度延缓了其通过肾小管的时间,加重了细胞损伤。水化和减少对比剂用量可有效预防CIAKI。
Abstract:
-

参考文献/References

[1] Bartorelli AL, Marenzi G. Contrast-induced nephropathy[J]. J Interv Cardiol, 2008, 21(1):74-85.
[2] Chronopoulos A, Cruz DN, Ronco C. Hospital-acquired acute kidney injury in the elderly [J]. Nat Rev Nephrol, 2010, 6(3):141-149.
[3] Ohno I, Hayashi H, Aonuma K, et al. Guidelines on the use of iodinated contrast media in patients with kidney disease 2012: digest version: JSN, JRS, and JCS Joint Working Group[J]. Clin Exp Nephrol, 2013, 17(4):441-479.
[4] Seeliger E, Sendeski M, Rihal CS, et al.Contrast-induced kidney injury: mechanisms, risk factors, and prevention[J].Eur Heart J, 33(16):2007-2015.
[5] Holmes D. Acute kidney injury: Hydration status linked to risk of contrast-induced AKI[J]. Nat Rev Nephrol, 2014, 10(5):241.
[6] Sadat U. Radiographic contrast-media-induced acute kidney injury: pathophysiology and prophylactic strategies[J]. ISRN Radiol, 2013, 2013:496438.
[7] Pallone TL, Zhang Z, Rhinehart K. Physiology of the renal medullary microcirculation[J]. Am J Physiol Renal Physiol, 2003, 284(2):F253-266.
[8] Sendeski MM. Pathophysiology of renal tissue damage by iodinated contrast media[J]. Clin Exp Pharmacol Physiol, 2011, 38(5):292-299.
[9] Sendeski M, Patzak A, Pallone TL, et al. Iodixanol, constriction of medullary descending vasa recta, and risk for contrast medium-induced nephropathy[J]. Radiology, 2009, 251(3):697-704.
[10] Sendeski MM, Persson AB, Liu ZZ, et al. Iodinated contrast media cause endothelial damage leading to vasoconstriction of human and rat vasa recta[J]. Am J Physiol Renal Physiol, 2012, 303(12):F1592-1598.
[11] Ulas T, Buyukhatipoglu H, Dal MS, et al. Urotensin-Ⅱ and endothelin-Ⅰ levels after contrast media administration in patients undergoing percutaneous coronary interventions[J]. J Res Med Sci, 2013, 18(3):205-209.
[12] Liu ZZ, Schmerbach K, Lu Y, et al. Iodinated contrast media cause direct tubular cell damage, leading to oxidative stress, low nitric oxide, and impairment of tubuloglomerular feedback[J]. Am J Physiol Renal Physiol, 2014, 306(8):F864-872.
[13] Curtis LM, Agarwal A. HOpe for contrast-induced acute kidney injury[J]. Kidney Int, 2007, 72(8):907-909.
[14] Donadio C, Tramonti G, Lucchesi A, et al. Tubular toxicity is the main renal effect of contrast media[J]. Ren Fail, 1996, 18(4):647-656.
[15] Calvin AD, Misra S, Pflueger A. Contrast-induced acute kidney injury and diabetic nephropathy[J]. Nat Rev Nephrol, 2010, 6(11):679-688.
[16] Carlson JE, Hedlund LJ, Trenkner SW, et al. Safety considerations in the power injection of contrast media via central venous catheters during computed tomographic examinations[J]. Invest Radiol, 1992, 27(5):337-340.
[17] Jost G, Lengsfeld P, Lenhard DC, et al. Viscosity of iodinated contrast agents during renal excretion[J]. Eur J Radiol, 2011, 80(2):373-377.
[18] Seeliger E, Becker K, Ladwig M, et al.Up to 50-fold increase in urine viscosity with isoosmolar contrast media in the rat[J]. Radiology, 2010, 256(2): 406-414.
[19] Hörl WH. Contrast induced nephropathy [J]. Wien Klin Wochenschr, 2009, 121(1-2):15-32.
[20] Arana E, Catalá-López F. Contrast-induced nephropathy in patients at risk of renal failure undergoing computed tomography: systematic review and meta-analysis of randomized controlled trials [J]. Med Clin(Barc), 2010, 135(8):343-350.
[21] Andreucci M, Solomon R, Tasanarong A. Side effects of radiographic contrast media: pathogenesis, risk factors, and prevention[J]. Biomed Res Int, 2014,2014:741018.
[22] Van Praet JT, De Vriese AS. Prevention of contrast-induced nephropathy: a critical review[J]. Curr Opin Nephrol Hypertens, 2007, 16(4):336-347.
[23] Zhang JJ, Hogstrom B, Malinak J, et al. Effects of viscosity on power and hand injection of iso-osmolar iodinated contrast media through thin catheters[J]. Acta Radiol, 2016, 57(5):557-564.
[24] 路新磊. 对比剂所致急性肾功能损害的预防策略[J]. 国际心血管病杂志, 2010, 37(3): 158-161.
[25] 包丽雯. 对比剂肾病预防措施研究[J]. 国际心血管病杂志, 2010, 37(5): 298-318.
[26] Stacul F, van der Molen AJ, Reimer P, et al. Contrast induced nephropathy: updated ESUR Contrast Media Safety Committee guidelines[J]. Eur Radiol, 2011, 21(12):2527-2541.
[27] Seeliger E, Ladwig M, Sargsyan L, et al. Proof of principle: hydration by low-osmolar mannitol-glucose solution alleviates undesirable renal effects of an iso-osmolar contrast medium in rats[J]. Invest Radiol, 2012, 47(4):240-246.
[28] Owen RJ, Hiremath S, Myers A, et al. Canadian Association of Radiologists consensus guidelines for the prevention of contrast-induced nephropathy: update 2012[J]. Can Assoc Radiol J, 2014, 65(2):96-105.
[29] Kooiman J, Seth M, Share D, et al. The association between contrast dose and renal complications post PCI across the continuum of procedural estimated risk[J]. PloS One,2014,9(3):e90233.

备注/Memo

备注/Memo:
通信作者:陈纪言,Email:chenjiyandr@126.com
更新日期/Last Update: 2016-09-20