索引超出了数组界限。 文章摘要
|本期目录/Table of Contents|

[1]王赛华,赵志宏,李新明.冠状动脉易损斑块及斑块易损性研究进展[J].国际心血管病杂志,2016,04:196-199.
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冠状动脉易损斑块及斑块易损性研究进展(PDF)

《国际心血管病杂志》[ISSN:1006-6977/CN:61-1281/TN]

期数:
2016年04期
页码:
196-199
栏目:
综述
出版日期:
2016-07-20

文章信息/Info

Title:
-
作者:
王赛华赵志宏李新明
201318 上海市浦东新区周浦医院心内科
Author(s):
-
关键词:
冠状动脉粥样硬化易损斑块斑块易损性
Keywords:
-
分类号:
-
DOI:
10.3969/j.issn.1673-6583.2016.04.002
文献标识码:
-
摘要:
易损斑块的形态学特征如微钙化、胆固醇结晶,局部血流动力学因素如内皮切应力、血管壁压力在易损斑块的形成与破裂中均起着重要的作用。了解易损斑块的微结构和分子学特征有助于识别易损斑块的破裂倾向。
Abstract:
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参考文献/References

[1] Tian J, Dauerman H, Toma C, et al. Prevalence and characteristics of tcfa and degree of coronary artery stenosis: An oct, ivus, and angiographic study[J]. J Am Coll Cardiol, 2014, 64(7):672-680.
[2] Falk E, Nakano M, Bentzon JF, et al. Update on acute coronary syndromes: The pathologists' view[J]. Eur Heart J, 2013, 34(10):719-728.
[3] Sluimer JC, Kolodgie FD, Bijnens AP, et al. Thin-walled microvessels in human coronary atherosclerotic plaques show incomplete endothelial junctions relevance of compromised structural integrity for intraplaque microvascular leakage[J]. J Am Coll Cardiol, 2009, 53(17):1517-1527.
[4] 郗 扬. 斑块内新生血管的作用及应对[J]. 国际心血管病杂志, 2014, 41(1):14-17.
[5] Mavrogeni S, Markousis-Mavrogenis G, Kolovou G. Contribution of cardiovascular magnetic resonance in the evaluation of coronary arteries[J]. World J Cardiol, 2014, 6(10):1060-1066.
[6] Tian J, Ren X, Vergallo R, et al. Distinct morphological features of ruptured culprit plaque for acute coronary events compared to those with silent rupture and thin-cap fibroatheroma: A combined optical coherence tomography and intravascular ultrasound study[J]. J Am Coll Cardiol, 2014, 63(21):2209-2216.
[7] Bentzon JF, Otsuka F, Virmani R, et al. Mechanisms of plaque formation and rupture[J]. Circ Res, 2014, 114(12):1852-1866.
[8] Chatzizisis YS, Antoniadis AP, Wentzel JJ, et al. Vulnerable plaque: The biomechanics of matter[J]. Atherosclerosis, 2014, 236(2):351-352.
[9] Pedrigi RM, de Silva R, Bovens SM, et al. Thin-cap fibroatheroma rupture is associated with a fine interplay of shear and wall stress[J]. Arterioscler Thromb Vasc Biol, 2014, 34(10):2224-2231.
[10] Virmani R, Kolodgie FD, Burke AP, et al. Lessons from sudden coronary death: A comprehensive morphological classification scheme for atherosclerotic lesions[J]. Arterioscler Thromb Vasc Biol, 2000, 20(5):1262-1275.
[11] Koskinas KC, Chatzizisis YS, Papafaklis MI, et al. Synergistic effect of local endothelial shear stress and systemic hypercholesterolemia on coronary atherosclerotic plaque progression and composition in pigs[J]. Int J Cardiol, 2013, 169(6):394-401.
[12] Stone PH, Saito S, Takahashi S, et al. Prediction of progression of coronary artery disease and clinical outcomes using vascular profiling of endothelial shear stress and arterial plaque characteristics: The prediction study[J]. Circulation, 2012, 126(2):172-181.
[13] Tousoulis D, Papageorgiou N, Synetos A, et al. Assessing vulnerable plaque: Is shear stress enough?[J]. Int J Cardiol, 2014, 172(1):e135-e138.
[14] Zhou J, Li YS, Chien S. Shear stress-initiated signaling and its regulation of endothelial function[J]. Arterioscler Thromb Vasc Biol, 2014, 34(10):2191-2198.
[15] Hecht HS. Coronary artery calcium scanning: Past, present, and future[J]. JACC Cardiovasc Imaging, 2015, 8(5):579-596.
[16] Alluri K, Joshi PH, Henry TS, et al. Scoring of coronary artery calcium scans: history, assumptions, current limitations, and future directions[J]. Atherosclerosis, 2015, 239(1):109-117.
[17] Hecht HS. Coronary artery calcium scanning: The key to the primary prevention of coronary artery disease[J]. Endocrinol Metab Clin North Am, 2014, 43(4):893-911.
[18] Mintz GS. Intravascular imaging of coronary calcification and its clinical implications[J]. JACC Cardiovasc Imaging, 2015, 8(4):461-471.
[19] Kataoka Y, Puri R, Hammadah M, et al. Spotty calcification and plaque vulnerability in vivo: Frequency-domain optical coherence tomography analysis[J]. Cardiovasc Diagn Ther, 2014, 4(6):460-469.
[20] Saia F, Komukai K, Capodanno D, et al. Eroded versus ruptured plaques at the culprit site of STEMI: In vivo pathophysiological features and response to primary PCI[J]. JACC Cardiovasc Imaging, 2015, 8(5):566-575.
[21] Prati F, Uemura S, Souteyrand G, et al. Oct-based diagnosis and management of stemi associated with intact fibrous cap[J]. JACC Cardiovasc Imaging, 2013, 6(3):283-287.
[22] Hu S, Jia H, Vergallo R, et al. Plaque erosion: In vivo diagnosis and treatment guided by optical coherence tomography[J]. JACC Cardiovasc Interv, 2014, 7(6):e63-64.
[23] Arbab-Zadeh A, Fuster V. The myth of the "vulnerable plaque": Transitioning from a focus on individual lesions to atherosclerotic disease burden for coronary artery disease risk assessment[J]. J Am Coll Cardiol, 2015, 65(8):846-855.
[24] Albaghdadi MS, Muse ED. Vulnerable plaque: Absence of evidence or evidence of absence[J]. J Am Coll Cardiol, 2015, 66(6):757-758.
[25] Sinclair H, Bourantas C, Bagnall A, et al. Oct for the identification of vulnerable plaque in acute coronary syndrome[J]. JACC Cardiovasc Imaging, 2015, 8(2):198-209.
[26] Toutouzas K, Tsiamis E, Karanasos A, et al. Morphological characteristics of culprit atheromatic plaque are associated with coronary flow after thrombolytic therapy: New implications of optical coherence tomography from a multicenter study[J]. JACC Cardiovasc Interv, 2010, 3(5):507-514.
[27] Puri R, Madder RD, Madden SP, et al. Near-infrared spectroscopy enhances intravascular ultrasound assessment of vulnerable coronary plaque: A combined pathological and in vivo study[J]. Arterioscler Thromb Vasc Biol, 2015, 35(11):2423-2431.
[28] Oemrawsingh RM, Cheng JM, Garcia-Garcia HM, et al. Near-infrared spectroscopy predicts cardiovascular outcome in patients with coronary artery disease[J]. J Am Coll Cardiol, 2014, 64(23):2510-2518.

备注/Memo

备注/Memo:
基金项目:上海市医学重点专科建设计划项目基金(ZK2015A17); 上海市浦东新区卫生和计划生育委员会重点学科建设基金资助(PDZx2014-01); 上海市浦东新区周浦医院重中之重建设专项基金(ZP2015A-01) 作者单位:201318 上海市浦东新区周浦医院心内科 通信作者:赵志宏,Email: zhihong_zhao@126.com
更新日期/Last Update: 2016-07-20