索引超出了数组界限。
[1] Peoples JN, Saraf A, Ghazal N, et al. Mitochondrial dysfunction and oxidative stress in heart disease[J]. Exp Mol Med, 2019, 51(12):1-13.
[2] Wilcox NS, Amit U, Reibel JB, et al. Cardiovascular disease and cancer: shared risk factors and mechanisms[J/OL]. Nat Rev Cardiol, 2024[2024-04-20]. https://pubmed.ncbi.nlm.nih. gov/38600368/.
[3] Yusuf S, Joseph P, Rangarajan S, et al. Modifiable risk factors, cardiovascular disease, and mortality in 155 722 individuals from 21 high-income, middle-income, and low-income countries (PURE): a prospective cohort study[J]. Lancet, 2020, 395(10226):795-808.
[4] Pi?eiro DJ, Codato E, Mwangi J, et al. Accelerated reduction in global cardiovascular disease is essential to achieve the Sustainable Development Goals[J]. Nat Rev Cardiol, 2023, 20(9):577-578.
[5] Redfern J, Tu Q, Hyun K, et al. Mobile phone text messaging for medication adherence in secondary prevention of cardiovascular disease[J]. Cochrane Database Syst Rev, 2024, 3(3):CD011851.
[6] Liu Y, Huang YJ, Xu C, et al. Mitochondrial dysfunction and therapeutic perspectives in cardiovascular diseases[J]. Int J Mol Sci, 2022, 23(24):16053.
[7] XiaD, Liu Y, Wu P, et al. Current advances of mitochondrial dysfunction and cardiovascular disease and promising therapeutic strategies[J]. Am J Pathol, 2023, 193(10):1485-1500.
[8] Tahrir FG, Langford D, Amini S, et al. Mitochondrial quality control in cardiac cells: mechanisms and role in cardiac cell injury and disease[J]. J Cell Physiol, 2019, 234(6):8122-8133.
[9] Zhao T, Zhang JJ, Lei H, et al. NRF1-mediated mitochondrial biogenesis antagonizes innate antiviral immunity[J]. EMBO J, 2023, 42(16):e113258.
[10] Dorn GW2nd, Vega RB, Kelly DP. Mitochondrial biogenesis and dynamics in the developing and diseased heart[J]. Genes Dev, 2015, 29(19):1981-1991.
[11] Vega RB, Horton JL, Kelly DP. Maintaining ancient organelles:mitochondrial biogenesis and maturation[J]. Circ Res, 2015, 116(11):1820-1834.
[12] Popov LD. Mitochondrial biogenesis: an update[J]. J Cell Mol Med, 2020, 24(9):4892-4899.
[13] Bouchez C, Devin A. Mitochondrial biogenesis and mitochondrial reactive oxygen species (ROS): a complex relationship regulated by the cAMP/PKA signaling pathway[J]. Cells, 2019, 8(4):287.
[14] Wu Z, Puigserver P, Andersson U, et al. Mechanisms controlling mitochondrial biogenesis and respiration through the thermogenic coactivator PGC-1[J]. Cell, 1999, 98(1):115-124.
[15] Malik N, Ferreira BI, Hollstein PE, et al. Induction of lysosomal and mitochondrial biogenesis by AMPK phosphorylation of FNIP1[J]. Science, 2023, 380(6642):eabj5559.
[16] Bj?rkegren JLM, Lusis AJ. Atherosclerosis: recent developments[J]. Cell, 2022, 185(10):1630-1645.
[17] Li AQ, Gao M, Liu BL, et al. Mitochondrial autophagy:molecular mechanisms and implications for cardiovascular disease[J]. Cell Death Dis, 2022, 13(5):444.
[18] Vyas HS, Jadeja RN, Vohra A, et al. CORM-A1 alleviates pro-atherogenic manifestations via miR-34a-5p downregulation and an improved mitochondrial function[J]. Antioxidants (Basel), 2023, 12(5):997.
[19] Sazonova MA, Sinyov VV, Ryzhkova AI, et al. Role of mitochondrial genome mutations in pathogenesis of carotid atherosclerosis[J]. Oxid Med Cell Longev, 2017, 2017:6934394.
[20] Li P, Xie CL, Zhong JK, et al. Melatonin attenuates ox-LDL-induced endothelial dysfunction by reducing ER stress and inhibiting JNK/Mff signaling[J]. Oxid Med Cell Longev, 2021, 2021:5589612.
[21] Ala M, Eftekhar SP. Target sestrin2 to rescue the damaged organ:mechanistic insight into its function[J]. Oxid Med Cell Longev, 2021, 2021:8790369.
[22] Pedriali G, Ramaccini D, Bouhamida E, et al. Perspectives on mitochondrial relevance in cardiac ischemia/reperfusion injury[J]. Front Cell Dev Biol, 2022, 10:1082095.
[23] Kwong JQ, Davis J, Baines CP, et al. Genetic deletion of the mitochondrial phosphate carrier desensitizes the mitochondrial permeability transition pore and causes cardiomyopathy[J]. Cell Death Differ, 2014, 21(8):1209-1217.
[24] Qi X, Wang J. Melatonin improves mitochondrial biogenesis through the AMPK/PGC1α pathway to attenuate ischemia/reperfusion-induced myocardial damage[J]. Aging(Albany NY), 2020, 12(8):7299-7312.
[25] Sun L, Zhao M, Yu XJ, et al. Cardioprotection by acetylcholine:a novel mechanism via mitochondrial biogenesis and function involving the PGC-1α pathway[J]. J Cell Physiol, 2013, 228(6):1238-1248.
[26] Vásquez-Trincado C, García-Carvajal I, Pennanen C, et al. Mitochondrial dynamics, mitophagy and cardiovascular disease[J]. J Physiol, 2016, 594(3):509-525.
[27] Yue P, Zhang Y, Liu L, et al. Yap1modulates cardiomyocyte hypertrophy via impaired mitochondrial biogenesis in response to chronic mechanical stress overload[J]. Theranostics, 2022, 12(16):7009-7031.
[28] Yu HJ, Zhang F, Yan PY, et al. LARP7 protects against heart failure by enhancing mitochondrial biogenesis[J]. Circulation, 2021, 143(20):2007-2022.
[29] Packer M. SGLT2 inhibitors: role in protective reprogramming of cardiac nutrient transport and metabolism[J]. Nat Rev Cardiol, 2023, 20(7):443-462.
[30] Sanches-Silva A, Testai L, Nabavi SF, et al. Therapeutic potential of polyphenols in cardiovascular diseases: regulation of mTOR signaling pathway[J]. Pharmacol Res, 2020, 152:104626.
[31] Horvath O, Ordog K, Bruszt K, et al. BGP-15 protects against heart failure by enhanced mitochondrial biogenesis and decreased fibrotic remodelling in spontaneously hypertensive rats[J]. Oxid Med Cell Longev, 2021, 2021:1250858.
[32] Chao YM, Wu KLH, Tsai PC, et al. Anomalous AMPK-regulated angiotensin AT1R expression and SIRT1-mediated mitochondrial biogenesis at RVLM in hypertension programming of offspring to maternal high fructose exposure[J]. J Biomed Sci, 2020, 27(1):68.
[33] Oller J, Gabandé-Rodríguez E, Ruiz-Rodríguez MJ, et al. Extracellular tuning of mitochondrial respiration leads to aortic aneurysm[J]. Circulation, 2021, 143(21):2091-2109.
[34] Xu S, Tao H, Cao W, et al. Ketogenic diets inhibit mitochondrial biogenesis and induce cardiac fibrosis[J]. Signal Transduct Target Ther, 2021, 6(1):54.
[35] Fan SW, Hu YH, You YP, et al. Role of resveratrol in inhibiting pathological cardiac remodeling[J]. Front Pharmacol, 2022, 13:924473.