索引超出了数组界限。
[1] Hopfner KP, Hornung V. Molecular mechanisms and cellular functions of cGAS-STING signalling[J]. Nat Rev Mol Cell Biol, 2020, 21(9):501-521.
[2] Li Q, Cao YZ, Dang C, et al. Inhibition of double-strand DNA-sensing cGAS ameliorates brain injury after ischemic stroke[J]. EMBO Mol Med, 2020, 12(4):e11002.
[3] Cao DJ, Schiattarella GG, Villalobos E, et al. Cytosolic DNA sensing promotes macrophage transformation and governs myocardial ischemic injury[J]. Circulation, 2018, 137(24):2613-2634.
[4] Li JK, Song ZP, Hou XZ. Scutellarin ameliorates ischemia/reperfusion injury-induced cardiomyocyte apoptosis and cardiac dysfunction via inhibition of the cGAS-STING pathway[J]. Exp Ther Med, 2023, 25(4):155.
[5] Xiong YH, Leng Y, Tian H, et al. Decreased MFN2 activates the cGAS-STING pathway in diabetic myocardial ischaemia-reperfusion by triggering the release of mitochondrial DNA[J]. Cell Commun Signal, 2023, 21(1):192.
[6] Feng YS, Imam Aliagan A, Tombo N, et al. Mitofilin heterozygote mice display an increase in myocardial injury and inflammation after ischemia/reperfusion[J]. Antioxidants (Basel), 2023, 12(4):921.
[7] Luecke S, Holleufer A, Christensen MH, et al. cGAS is activated by DNA in a length-dependent manner[J]. EMBO Rep, 2017, 18(10):1707-1715.
[8] Srikanth S, Woo JS, Wu BB, et al. The Ca2+ sensor STIM1 regulates the typeⅠ interferon response by retaining the signaling adaptor STING at the endoplasmic reticulum[J]. Nat Immunol, 2019, 20(2):152-162.
[9] Dobbs N, Burnaevskiy N, Chen DD, et al. STING activation by translocation from the ER is associated with infection and autoinflammatory disease[J]. Cell Host Microbe, 2015, 18(2):157-168.
[10] Haag SM, Gulen MF, Reymond L, et al. Targeting STING with covalent small-molecule inhibitors[J]. Nature, 2018, 559(7713):269-273.
[11] Mukai K, Konno H, Akiba T, et al. Activation of STING requires palmitoylation at the Golgi[J]. Nat Commun, 2016, 7:11932.
[12] Zhang RX, Kang R, Tang DL. The STING1 network regulates autophagy and cell death[J]. Signal Transduct Target Ther, 2021, 6(1):208.
[13] Murthy AMV, Robinson N, Kumar S. Crosstalk between cGAS-STING signaling and cell death[J]. Cell Death Differ, 2020, 27(11):2989-3003.
[14] Ali Pour P, Hosseinian S, Kheradvar A. Mitochondrial transplantation in cardiomyocytes: foundation, methods, and outcomes[J]. Am J Physiol Cell Physiol, 2021, 321(3):C489-C503.
[15] Peng C, Zhang YX, Lang XY, et al. Role of mitochondrial metabolic disorder and immune infiltration in diabetic cardiomyopathy: new insights from bioinformatics analysis[J]. J Transl Med, 2023, 21(1):66.
[16] Teixeira RB, Pfeiffer M, Zhang P, et al. Reduction in mitochondrial ROS improves oxidative phosphorylation and provides resilience to coronary endothelium in non-reperfused myocardial infarction[J]. Basic Res Cardiol, 2023, 118(1):3.
[17] Algoet M, Janssens S, Himmelreich U, et al. Myocardial ischemia-reperfusion injury and the influence of inflammation[J]. Trends Cardiovasc Med, 2023, 33(6):357-366.
[18] Zhang L, Cui TX, Wang XJ. The interplay between autophagy and regulated necrosis[J]. Antioxid Redox Signal, 2023, 38(7/9):550-580.
[19] Hassan S, Cecen B, Pe?a-Garcia R, et al. Survival and proliferation under severely hypoxic microenvironments using cell-laden oxygenating hydrogels[J]. J Funct Biomater, 2021, 12(2):30.
[20] Heusch G. Myocardial ischaemia-reperfusion injury and cardioprotection in perspective[J]. Nat Rev Cardiol, 2020, 17(12):773-789.
[21] Zhang XQ, Qu HY, Yang T, et al. Astragaloside Ⅳ attenuate MI-induced myocardial fibrosis and cardiac remodeling by inhibiting ROS/caspase-1/GSDMD signaling pathway[J]. Cell Cycle, 2022, 21(21):2309-2322.
[22] Powers KA, Szászi K, Khadaroo RG, et al. Oxidative stress generated by hemorrhagic shock recruits Toll-like receptor 4 to the plasma membrane in macrophages[J]. J Exp Med, 2006, 203(8):1951-1961.
[23] Rosenfeld MA, Yurina LV, Vasilyeva AD. Antioxidant role of methionine-containing intra- and extracellular proteins[J]. Biophys Rev, 2023, 15(3):367-383.
[24] Wu H, Liu QH, Yang NX, et al. Polystyrene-microplastics and DEHP co-exposure induced DNA damage, cell cycle arrest and necroptosis of ovarian granulosa cells in mice by promoting ROS production[J]. Sci Total Environ, 2023, 871:161962.
[25] 周逸然, 赵鹏军. 线粒体功能损伤对心肌细胞的影响[J]. 国际心血管病杂志, 2023, 50(5):295-297.
[26] King KR, Aguirre AD, Ye YX, et al. IRF3 and typeⅠ interferons fuel a fatal response to myocardial infarction[J]. Nat Med, 2017, 23(12):1481-1487.
[27] Ter Horst EN, Krijnen PAJ, Hakimzadeh N, et al. Elevated monocyte-specific typeⅠ interferon signalling correlates positively with cardiac healing in myocardial infarct patients but interferon alpha application deteriorates myocardial healing in rats[J]. Basic Res Cardiol, 2018, 114(1):1.
[28] Hu SY, Gao Y, Gao RF, et al. The selective STING inhibitor H-151 preserves myocardial function and ameliorates cardiac fibrosis in murine myocardial infarction[J]. Int Immunopharmacol, 2022, 107:108658.
[29] Hao F. An overview of the crosstalk between YAP and cGAS-STING signaling in non-small cell lung cancer: it takes two to tango[J]. Clin Transl Oncol, 2022, 24(9):1661-1672.
[30] Xu DW, Tian YZ, Xia Q, et al. The cGAS-STING pathway:
novel perspectives in liver diseases[J]. Front Immunol, 2021, 12:682736.
[31] Wan DS, Jiang W, Hao JW. Research advances in how the cGAS-STING pathway controls the cellular inflammatory response[J]. Front Immunol, 2020, 11:615.
[32] Ma RH, Ortiz Serrano TP, Davis J, et al. The cGAS-STING pathway: the role of self-DNA sensing in inflammatory lung disease[J]. FASEB J, 2020, 34(10):13156-13170.
[33] Zhang DW, He Y, Ye XD, et al. Activation of autophagy inhibits nucleotide-binding oligomerization domain-like receptor protein 3 inflammasome activation and attenuates myocardial ischemia-reperfusion injury in diabetic rats[J]. J Diabetes Investig, 2020, 11(5):1126-1136.
[34] Cai Y, Ying F, Liu H, et al. Deletion of rap1 protects against myocardial ischemia/reperfusion injury through suppressing cell apoptosis via activation of STAT3 signaling[J]. FASEB J, 2020, 34(3):4482-4496.
[35] Frangogiannis NG. Cell biological mechanisms in regulation of the post-infarction inflammatory response[J]. Curr Opin Physiol, 2018, 1:7-13.
[36] Cai X, Chiu YH, Chen ZJ. The cGAS-cGAMP-STING pathway of cytosolic DNA sensing and signaling[J]. Mol Cell, 2014, 54(2):289-296.
[37] Rech L, Abdellatif M, P?ttler M, et al. Small molecule STING inhibition improves myocardial infarction remodeling[J]. Life Sci, 2022, 291:120263.
[38] Zhang Y, Chen WZ, Wang Y. STING is an essential regulator of heart inflammation and fibrosis in mice with pathological cardiac hypertrophy via endoplasmic reticulum (ER) stress[J]. Biomed Pharmacother, 2020, 125:110022.
[39] Martins-Marques T, Hausenloy DJ, Sluijter JPG, et al. Intercellular communication in the heart: therapeutic opportunities for cardiac ischemia[J]. Trends Mol Med, 2021, 27(3):248-262.
[40] Zhang C, Hao H, Wang YS, et al. Intercellular mitochondrial component transfer triggers ischemic cardiac fibrosis[J]. Sci Bull (Beijing), 2023, 68(16):1784-1799.