索引超出了数组界限。
[1] O’Gara PT, Kushner FG, Ascheim DD, et al. 2013 ACCF/AHA guideline for the management of ST-elevation myocardial infarction: a report of the American College of Cardiology Foundation/American Heart Association task force on practice guidelines[J]. Circulation, 2013, 127(4):e362-e425.
[2] Authors/Task Force members, Windecker S, Kolh P, et al. 2014 ESC/EACTS guidelines on myocardial revascularization:the task force on myocardial revascularization of the European Society of Cardiology(ESC)and the European Association for Cardio-Thoracic Surgery(EACTS)developed with the special contribution of the European Association of Percutaneous Cardiovascular Interventions(EAPCI)[J]. Eur Heart J, 2014, 35(37):2541-2619.
[3] Steg PG, Thuaire C, Himbert D, et al. DECOPI(DEsobstruction COronaire en Post-Infarctus): a randomized multi-centre trial of occluded artery angioplasty after acute myocardial infarction[J].Eur Heart J, 2004, 25(24):2187-2194.
[4] Erne P, Schoenenberger AW, Burckhardt D, et al. Effects of percutaneous coronary interventions in silent ischemia after myocardial infarction: the SWISSI Ⅱ randomized controlled trial[J]. JAMA, 2007, 297(18):1985-1991.
[5] Silva JC, Rochitte CE, Júnior JS, et al. Late coronary artery recanalization effects on left ventricular remodelling and contractility by magnetic resonance imaging[J]. Eur Heart J, 2005, 26(1):36-43.
[6] Fan Y, Bai X, Chen Y, et al. Late percutaneous coronary intervention prevents left ventricular remodeling and improves clinical outcomes in patients with ST-elevation myocardial infarction[J]. Clin Cardiol, 2015, 38(2):82-91.
[7] Centurión OA. The open artery hypothesis: beneficial effects and long-term prognostic importance of patency of the infarct-related coronary artery[J]. Angiology, 2007, 58(1):34-44.
[8] Braunwald E, Rutherford JD. Limitation of infarct size and the open artery hypothesis: a conversation with Eugene Braunwald, MD[J]. Circulation, 2016, 134(12):839-846.
[9] Sabia PJ, Powers ER, Ragosta M, et al. An association between collateral blood flow and myocardial viability in patients with recent myocardial infarction[J]. N Engl J Med, 1992, 327(26):1825-1831.
[10] Goel PK, Bhatia T, Kapoor A, et al. Left ventricular remodeling after late revascularization correlates with baseline viability[J]. Tex Heart Inst J, 2014, 41(4):381-388.
[11] Maek LA, Silva JC, Bellenger NG, et al. Late percutaneous coronary intervention for an occluded infarct-related artery in patients with preserved infarct zone viability: a pooled analysis of cardiovascular magnetic resonance studies[J]. Cardiol J, 2013, 20(5):552-559.
[12] Nakagawa M, Takemura G, Kanamori H, et al. Mechanisms by which late coronary reperfusion mitigates postinfarction cardiac remodeling[J]. Circ Res, 2008, 103(1):98-106.
[13] Li Y, Takemura G, Kosai K, et al. Critical roles for the Fas/Fas ligand system in postinfarction ventricular remodeling and heart failure[J]. Circ Res, 2004, 95(6):627-636.
[14] Kloner RA, Hwang H. New insights into the open artery hypothesis[J]. Circ Res, 2008, 103(1):1-3.
[15] Hochman JS, Lamas GA, Buller CE, et al. Coronary intervention for persistent occlusion after myocardial infarction[J]. N Engl J Med, 2006, 355(23):2395-2407.
[16] Hochman JS, Sleeper LA, White HD, et al. One-year survival following early revascularization for cardiogenic shock[J]. JAMA, 2001, 285(2):190-192.
[17] Buccheri S, Capranzano P, Condorelli A, et al. Risk stratification after ST-segment elevation myocardial infarction[J]. Expert Rev Cardiovasc Ther, 2016, 14(12):1349-1360.
[18] Zhang D, Li Y, Yin D, et al. Risk stratification of periprocedural myocardial infarction after percutaneous coronary intervention: analysis based on the SCAI definition[J]. Catheter Cardiovasc Interv, 2017, 89(S1):534-540.
[19] Suresh R, Li X, Chiriac A, et al. Transcriptome from circulating cells suggests dysregulated pathways associated with long-term recurrent events following first-time myocardial infarction[J]. J Mol Cell Cardiol, 2014, 74:13-21.
[20] Pfisterer ME, Buser P, Osswald S, et al. Time dependence of left ventricular recovery after delayed recanalization of an occluded infarct-related coronary artery: findings of a pilot study[J]. J Am Coll Cardiol, 1998, 32(1):97-102.
[21] Yip HK, Wu CJ, Yang CH, et al. Delayed post-myocardial infarction invasive measures, helpful or harmful? A subgroup analysis[J]. Chest, 2004, 126(1):38-46.
[22] Niccoli G, Burzotta F, Galiuto L, et al. Myocardial no-reflow in humans[J]. J Am Coll Cardiol, 2009, 54(4):281-292.
[23] Harrison RW, Aggarwal A, Ou FS, et al. Incidence and outcomes of no-reflow phenomenon during percutaneous coronary intervention among patients with acute myocardial infarction[J]. Am J Cardiol, 2013, 111(2):178-184.
[24] Yousef ZR, Marber MS, Redwood SR. Late opening of the infarct related artery: an open or shut case?[J]. Heart, 2005, 91(5):561-562.
[25] Fishbein MC, Maclean D, Maroko PR. Experimental myocardial infarction in the rat: qualitative and quantitative changes during pathologic evolution[J]. Am J Pathol, 1978, 90(1):57-70.